“This study shows that if an overweight person is able to maintain an initial weight loss – in this case for a year – the body will eventually ‘accept’ this new weight and thus not fight against it, as is otherwise normally the case when you are in a calorie-deficit state,”If an o/w person-or presumably any person including slims, 'cos they're people too-keeps starving themselves, after a year, their metabolic function may reset itself to that level of hunger.
The interesting uplifting news in this study is that if you are able to maintain your weight loss for a longer period of time, it seems as if you have ‘passed the critical point’, and after this point, it will actually become easier for you to maintain your weight loss than is was immediately after the initial weight loss.I beg to differ. Its the other way round. Adjust metabolic function (first) and that will alter outcomes insomuch as your body responds to any particular [metabolic] alteration, as I've been boring on about for it seems like aeons.
If she plays her cards right, Professor Signe Sørensen Torekov the head honcho of this study-she could be on course to be the second most famous Dane after himself.
Isn't this magnificent? No more enforced starvation for anyone; whoo hoo: Paaaaartay!!!
This particular study's topsy-turvey findings came by measuring the levels of two chemicals GLP-1 and PYY 3-36, said to be present, in the blood, when there's "appetite inhibition". The levels of both were measured after meals in people who had lost 13% of their weight. Three measurements were taken; before they'd lost the weight, just after their weight loss and 52 weeks after sustaining said weight loss.
These chemicals increased after the 52 week period to a level reflective of a lack of unwanted hunger. According to this study, the body finally "accept(s)" lowered intake, in theory of course. That's great.
It should be simple to confirm whether these pointers are simply present along with, or are hunger cessation in action. Find a way or ways to reduce/inhibit levels of GLP-1 and PYY 3-36 directly, stand back and watch people's hunger/appetite fall without any need for privation. It will be sensational-given the desperation to stave off the global adipocalypse.
Significantly altering hunger/metabolic is nothing new. Having dieting career or merely the threat of one hanging over you, can and does significantly adjust hunger/metabolic function. Yes, merely contemplating springing a diet on yourself day after day for years is enough to trigger some of the delightful adjustments of your metabolic function and nervous system.
Repeat, some people are going to discover that what they thought was a neurotic flaky nature is actually a side effect of long-term calorie restriction or the threat of it.
I altered mine back albeit accidentally. I barely altered in weight, tricky though it undoubtedly is, altering hunger seems way easier than altering the extent to which the body stores fat.
I'm sure I've said enough times to bore people almost as much as I'm bored with saying it. There's no way bodies can only gain weight. That nonsense has been put about solely via the calories in/out assertion. All that's lacking is the means of adjusting the metabolic mechanism to trigger or set that off reversal.
Each and every force must have an equal and countervailing force.
It's obvious in the way even that weight gain does not happen at the same speed. If your weight goes up one year and less or nothing the next. That's a downward adjustment, a theoretical reversal in terms of absence of gain. The body does this without pain, privation or punishment. Suggesting all that's necessary is making adjustments at source.
If what's missing is reducing the levels of a couple of chemicals, then vamos, let's go kiddos.
If this had been done in a timely fashion, 8 year old, Hana Tarraf wouldn't have had to have a gastric band fitted merely to try and arrest hypothalamically induced weight gain. The originator of this, a tumour, may not be so simple to permanently vanquish, but she could have been spared from the unnecessary anguish of agonising hyperphagia.
Hyperphagia-genuinely excessive/heightened hunger functioning is distressing enough to be relieved with or without weight loss. Never forget who's paying for this bizarre obsession with getting fat people and therefore everyone else, to starve themselves.
I hope this is helping you to see how that makes absolutely no sense whatsoever. There is no point I can see to it, but I'll stand correction. Feel free to explain why the cart must be put before the horse, instead of the right way round.
I don't give a damn where you are coming from on this, ask yourself; Why are ob researchers avoiding directly adjusting metabolic function?
Even more fascinating still; What is it are they avoiding?
That's more of a mystery than the plasticity of metabolic function.
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